Such entrapment strategies can allow for neutralization of host inflammation following viral infection, emphasizing the mechanistic similarities between exosomes and viruses and the potential to optimize exosome “nanodecoys.”52 Recent studies have also shown that type II alveolar epithelial cells in the lungs and goblet secretory cells in the nasal mucosa are specific cellular targets of the SARS‐CoV‐2 virus via binding of the viral spike (S‐) protein to angiotensin‐converting enzyme 2 (ACE2) on the cell surface membrane for viral entry.53 Here, ACE2 is linked to viral infectious disease.