Besides, in the population of COVID‐19 patients with chronic lung injuries, AT2 cells exhibit preexisting dysregulation of viral infection associated genes, including verified and putative entry receptors and priming proteases (ACE2 and putative BSG, NPR1, HSPA5, as entry receptors; and TMPRSS2, CTSL, or FURIN, as priming proteases), which will facilitate SARS‐CoV‐2 infection.130. Here, ACE2 is linked to COVID-19.