HOTAIR could promote glioblastoma cell cycle progression (Zhang et al., 2015); FOXM1-AS could enhance self-renewal and tumorigenesis of glioblastoma stem-like cells (Zhang et al., 2017); H19 could promote glioblastoma cell invasion, angiogenesis, and tube formation (Jia et al., 2016); MALAT1 could decrease the sensitivity of glioblastoma cells to TMZ (Li et al., 2017). Here, MALAT1 is linked to glioblastoma.