ALKBH5 and Miyoshi myopathy: Next, we restored TRAF1 expression in ALKBH5 KD MM cells to clarify whether TRAF1 was a significant contributor to the function of ALKBH5 in MM proliferation (Fig. S7K) and found that the growth inhibitory effects caused by ALKBH5 deficiency were largely rescued by restoration of TRAF1 expression (Figs. 7J–M; S7L–N).