Besides, Wang et al. reported that the expression of exosomal miR-107 derived from patients with AF was significantly higher than the expression from patients without AF.470 Incubation of ECs with AF-derived exosomes or miR-107 mimics significantly inhibits cell viability and migration, while enhancing cell apoptosis by regulating miR-107/USP14 pathway. The gene discussed is USP14; the disease is atrial fibrillation.