The polymorphic insertion of the HERV-K113 element, a full-length HERV-K HML-2 provirus, had a significantly higher prevalence in the genomes of specific patients of Sjögren’s syndrome, multiple sclerosis, systemic lupus erythematosus, and rheumatoid arthritis70,76. The gene discussed is CLEC10A; the disease is systemic lupus erythematosus.