For example, miR-29a could reverse circSTAT3-induced hepatoblastoma cell growth through targeting STAT3 [14]; Weiss et al. found downregulation of miR-483 could inhibit the proliferation of hepatoblastoma cells by suppressing the cell apoptosis [15]; downregulation of miR-203 could promote the angiogenesis of hepatoblastoma cells via activation of VEGFA [16]. This evidence concerns the gene VEGFA and hepatoblastoma.