Given that effector‐triggered immunity is generally sensitive to NLR dosage, and that overexpression of functional NLR genes can result in autoimmunity13, we wondered whether the resistance carried by the transgenic T2 progeny lines could result from autoimmunity triggered by overexpression of the Rps11-transgene under the regulation of the AtUbi3 promoter. Here, RPS11 is linked to Autoimmunity.