To mention are, among others, markers for inflammation (TNFR-1a, periostin), cardiomyocyte stretch (BNP), and angiogenesis (VGEFR, angiogenin), suggesting that cardiac remodeling, inflammation, and fibrosis are closely associated with each other in patients with HF and diabetes [89, 90]. Here, NPPB is linked to hydrops fetalis.