To determine the impact of the SUR1/Kir6.2 subtype on the etiology of AD, we analyzed the transcripts of each KATP channel component in datasets from human postmortem brains, with or without AD, obtained from the Gene Expression Ominibus (GEO) of the National Center for Biotechnology Information (NCBI). This evidence concerns the gene KCNJ11 and Alzheimer disease.