IGF2BP1 and COVID-19: We were particularly interested in IGF2BP1, which was among the few host factors that are common interactors to all three viruses, and was found to be able to enhance translation of hepatitis C virus RNAs (HCV) by binding to HCV UTRs.42 Moreover, we found that SARS-CoV-2 infection stimulates IGF2BP1 mRNA expression both in cell lines and the lungs of COVID-19 patients (Supplementary information, Fig. S4d, e).