TNNT1 and nemaline myopathy 5: Slow TnT mutations that cause deletion of the C-terminal 14 amino acids have been reported to cause ANM-like myopathies in compound heterozygotes with some of the above mutations (van der Pol et al., 2014; Petrucci et al., 2021), confirming the functional importance of the C-terminal end segment of TnT (Lopez Davila et al., 2020).