Aligned with our results, a phosphoproteomic dataset of PC-9 lung adenocarcinoma cells harboring active EGFR via E746-A750 activating deletions (PhosphoSitePlus; https://www.phosphosite.org/uniprotAccAction?id=Q9H8S9), showed elevated levels of MOB1 phosphorylated at Y95, supporting our findings that EGFR-MOB1-YAP/TAZ signaling may play an important role in cancers harboring EGFR alterations41,42. This evidence concerns the gene EGFR and lung adenocarcinoma.