SNHG8 and gastric cancer: Our data support that the acting mechanism of SNHG8 could be multiple: (1) it activated BCL-2, preventing GC cells from apoptosis; (2) it activated CCND1, regulating the cell cycle; and (3) it activated CDH1, CDH2Snai1, and VIM, enhancing the epithelial–mesenchymal transition, thus promoting the migration, invasion, and metastasis of GC cells.