ACE2 and infection: In contrast to HMVEC‐L with endogenous levels of ACE2, overexpression of ACE2 in HMVEC‐L resulted in active viral replication upon infection with either 2 × 106 PFUs of SARS‐CoV‐2 or 6 × 104 PFUs of SARS‐CoV‐2 (Figure 2i).