In addition, Because CRP can participate in the inflammation process by binding to ligands, reducing endothelial cell nitric oxide synthase activity by binding to CD32, and promoting the phagocytic function of macrophages by regulating effect, we believe that CRP, as a sensitive inflammatory marker involved in the occurrence and development of atherosclerosis, may play an important role in the formation of proximal coronary atherosclerosis in patients with the myocardial bridge [17, 18]. Here, CRP is linked to atherosclerosis.