The oncogenic features of miR-182 in ovarian cancer are noticeably due to impairment in double-strand breaks repair in DNA, negative control of BRCA1, suppression of metastasis suppressor 1 (MTSS1), and overexpression of high-mobility group AT-hook 2 (HMGA2) [155, 159–162]. This evidence concerns the gene MTSS1 and ovarian carcinoma.