However, in a previous work, unseparated MDSCs, which represent a mixture of CD11b+CD11c+ and CD11b+CD11c− MDSCs, did not abrogate the GVT effect in a parent into F1 BMT model (18), indicating that none of the MDSC subpopulations reduce the capacity of allogeneic T cells to attack residual tumor cells. The gene discussed is ITGAX; the disease is neoplasm.