This observation is in agreement with previous findings: Kwa et al. observed that 24% of GBS patients had IgG antibodies against non-myelinating human Schwann cells [36], and Vallat et al. also detected that a significant percentage of CIDP and GBS patients (about 25%) presented with IgG or IgM reactivity against myelin and that the staining patterns on Schwann cells were diverse, suggesting that diverse myelin antigens are being recognized by the autoantibodies [37]. This evidence concerns the gene CD40LG and Guillain-Barre syndrome.