NKG2A expression was furthermore associated with dividing CD8 T cells, suggesting that this particular TIL population represents a chronically activated subset of CD8 T cells in tumors and potentially marks the tumor‐reactive fraction, as was suggested for PD‐1high, TIM‐3 and CD39 positive T cells.42, 43, 44, 45. This evidence concerns the gene HAVCR2 and neoplasm.