To determine the potential of RvD2 to stimulate skeletal muscle regeneration in DMD, we first quantified the density of MuSC (Pax7+ cells) and differentiated myoblasts (Myog+ cells) in TA muscle of mdx mice treated with RvD2, prednisone, or saline (Fig. 5a–f, and Supplementary Fig. 10). The gene discussed is MYOG; the disease is Duchenne muscular dystrophy.