Several initiated signaling pathways, including linc00467/miR-20b-5p/cyclin D1 pathway [20] and STAT1-induced upregulation of linc00467 promoted lung adenocarcinoma progression through epigenetically suppressing dickkopf WNT signaling pathway inhibitor 1 to activate Wnt/β-catenin signaling [16]. Here, CCND1 is linked to lung adenocarcinoma.