Previous studies have demonstrated that linc00467 was abnormally expressed (most were upregulated, individual decreased) in various types of malignant tumors, which contribute to tumorigenesis through different kinds of signaling pathways, such as miR-9-5p/peroxisome proliferator activated receptor alpha, miR-20b-5p/cyclin D1, and DNA methyltransferase 1/p53 [18,20,21]. Here, LINC00467 is linked to cancer.