SLCO4A1 and gastric cancer: Then we found that overexpression of SLCO4A1-AS1 enhanced cell viability, inhibited apoptosis, and significantly accelerated cell migration and invasion in gastric cancer cells, while knockdown of SLCO4A1-AS1 had the opposite effect (P < 0.05, Figures 1(f), 1(g), and 2(a)–2(l)).