Our data show that this model is highly reproducible in terms of its response to TGFβ1, and replicates many of the molecular changes characteristic of IPF such as increases in pro-fibrotic gene expression, tissue and soluble collagens, and increased myofibroblast numbers (DePianto et al., 2015). Here, TGFB1 is linked to idiopathic pulmonary fibrosis.