CXCL13 level was increased in the serum of patients with sepsis, and CXCL13 also acts via p38MAPK signaling to drive LPS-induced hyperpermeability of the endothelium in human umbilical vein endothelial cells, suggesting that targeting CXCL13 may alleviate sepsis [45]. Here, CXCL13 is linked to Sepsis.