In the current study of women with PTL without IAI, we (1) characterized 91 DEPs and their potential biological pathways (i.e., LXR/RXR activation, APR signaling, FXR/RXR activation, coagulation system, and complement system) in pooled plasma samples of women with SPTB compared to those of women with TB, using quantitative label-free proteomic analysis; and (2) attempted to verify selected target proteins (8 DEPs) using clot-based and ELISA-based assays. The gene discussed is NR1H4; the disease is tuberculosis.