Our results showed that ITK expression was positively correlated with cancer suppressor gene CD244 (p = 9.1e−10) and SOCS1 (p = 4.8e−09) among patients with a favorable prognosis, suggesting that the function of ITK may be similar with CD244 and SOCS1. Moreover, oncogene SMARCD1 was upregulated in ovarian cells exposed to ascites, allowing a non-stimulatory effect on cancer cell migration [39]. The gene discussed is ITK; the disease is cancer.