IL17A and infection: Depleting CD90.2+ ILCs significantly reduced the inflammatory responses as assessed by ear thickness and pathology score two weeks post-infection in S. epidermidis colonized and L. major infected Rag1-/- mice compared to S. epidermidis colonized and L. major infected Rag1-/- mice that did not receive the anti-CD90.2 antibody (‘control’) (Fig 6H) To test that IL-17 production by ILCs was responsible for mediating the pathology in Rag1-/- mice, we treated mice with anti-IL-17A mAb.