MPO and lymphopenia: These include the release of arachidonic acid metabolites and platelet-aggravating factors, cytotoxic oxygen–derived free radicals [13], myeloperoxidase [14], elastase [15], various hydrolytic enzymes such as acid phosphatases [16], etc. The relative lymphopenia observed post TAVI can be explained by the endogenous cortisol segregation response to the procedure’s stress [17].