The evidence that NKG2C+CD57+ natural killer (NK) cell responses induced by CMV confer enhanced killing of virus-infected cells [38,39,40,41] and the association between NKG2C+CD57+ γδ T cells and CMV infection in our cohort prompted us to investigate the role of these cells in controlling viral replication in our Ugandan infant cohort. The gene discussed is KLRC2; the disease is cytomegalovirus infection.