The evidence that NKG2C+CD57+ natural killer (NK) cell responses induced by CMV confer enhanced killing of virus-infected cells [38,39,40,41] and the association between NKG2C+CD57+ γδ T cells and CMV infection in our cohort prompted us to investigate the role of these cells in controlling viral replication in our Ugandan infant cohort. This evidence concerns the gene B3GAT1 and cytomegalovirus infection.