For example, pulmonary infection by SARS-CoV-2 can cause systemic increases in pro-inflammatory cytokines such as IL-6, CRP, IL-2, IL-7, TNF-α, MCP-1 (monocyte chemoattractant+ protein 1), and G-CSF (granulocyte-colony-stimulating factor), and the increase in pro-inflammatory factors correlates with cardiac injury during infection [3]. Here, TNF is linked to infection.