Although anosmia is not particularly surprising for a respiratory virus, if, for example, it is assumed to result from direct viral damage to the olfactory bulb (Dey et al. 2020), it may be an important clinical symptom to consider in a patient without other upper-respiratory symptoms as a sign of dysregulated inflammatory response (Han et al. 2020), including potential dysregulation of ADAR editing. This evidence concerns the gene ADAR and Kallmann syndrome.