Almost immediately, we transfected with a miR-101a-3p mimic and inhibitor into NSCLC cells and determined the level of ATG4D, showing that the expression level of ATG4D was significantly downregulated by miR-101a-3p mimic treatment, whereas inhibition of miR-101a-3p upregulated ATG4D expression in NSCLC cells (Figure 3c). The gene discussed is ATG4D; the disease is non-small cell lung carcinoma.