METTL3 and myocardial infarction: In contrast, knock out of methyltransferase-like 3 (METTL3), a N6-methyladenosine writer, whose expression levels raise postnatally, induces cardiomyocyte cell cycle re-entry, reduces scar size and boosts cardiac function after myocardial infarction, by regulating the miR-143-YAP axis (224).