HIF1A and steatosis: Myeloid specific HIF1α stabilisation worsened steatosis and inflammation, with increased macrophage infiltration in the liver, higher expression of the proinflammatory cytokines macrophage chemoattractant protein 1 (MCP1) and interleukin (IL)-1b in liver macrophages, and higher hepatic levels of Mcp1 and tumour necrosis factor α (Tnfa) mRNA.