miR-302c, miR423-5p, miR-210, miR-376c, and miR-185 were found to be highly expressed in rapid-progressive IPF compared to slow-progressing IPF whereas miR processing components, Ago1 and Ago2, levels were lower in the rapid-progressive IPF compared to slow progressive IPF or control normal lungs [147,148]. Here, AGO1 is linked to idiopathic pulmonary fibrosis.