Enriched pathways, including PI3K-Akt signaling pathway, mitogen-activated protein kinase signaling pathway, focal adhesion, Wnt signaling pathway, Toll-like receptor signaling pathway, tumor growth factor-beta signaling pathway, and ECM-receptor interaction revealed that LINC01614 might exert potential regulatory effect in GC through multiple mechanisms (Figure 5D). This evidence concerns the gene AKT1 and gastric cancer.