The vast majority of Dravet syndrome cases are caused by a LoF mutation in one allele of the SCN1A gene, resulting in voltage-gated sodium channels with a non-functional NaV1.1 subunit primarily in inhibitory GABAergic neurons, leading to hyperexcitability and seizures associated with high risk of sudden infant death (Samanta, 2020). This evidence concerns the gene SCN1A and Dravet syndrome.