Takashi Mori et al. [18,22,26] reported that FA can reduce amyloid APP metabolism by reducing BACE1 expression and β-secretase activity in mice, thereby improving Aβ1-40 and Aβ1-42 deposition in the brain parenchyma and cerebral vessels of AD mice and slowing related toxic reactions. Here, BACE1 is linked to Alzheimer disease.