A recent study demonstrated that AUT2, a positive modulator of Kv3.1, restored an enhanced wave IV in the auditory brainstem response (ABR) recordings in Fmr1 KO mice, which may have suggested a therapeutic potential for sensory symptoms in FXS individuals, although the pharmacological actions for ABR rescue are likely through modulating the activity of MNTB neurons, not GABAergic interneurons [126]. The gene discussed is FMR1; the disease is fragile X syndrome.