In patients with allergic asthma, an imbalance in lymphocyte immunity, especially between Th1 and Th2 responses, leads to immunoglobulin E (IgE) production, T cell and eosinophil infiltration, airway hyperresponsiveness, mucus cell hyperplasia, and airway remodeling, resulting in the appearance of asthma symptoms [5]. The gene discussed is IGHE; the disease is asthma.