It inhibits the expression of Toll-like receptors (TLRs), macrophage inflammatory protein-1 alpha (MIP-1α), T-cell proliferation and the release of inflammatory cytokines such as interleukine-6 (IL-6), interleukine-8 (IL-8), and tumor necrosis factor alpha (TNFα), which have a key role in the development of uveitis [7,28,30,31,32]. This evidence concerns the gene CCL3 and uveitis.