Overexpression of dynamin-2 prior to viral infection led to a statistically significant increase in CXCL10 and CXCL8, while downregulating dynamin-2 led to statistically significant reductions in CCL2, CCL5, CXCL1, CXCL11, IL-1ra, IL-6, MIF, and Serpin E1 [99]. This evidence concerns the gene DNM2 and viral infectious disease.