The ionizing radiation-induced K-RAS/c-RAF/p38 pathway activation enhances cell invasion and migration ability in cervical cancer [31]; moreover, K-RAS or IR induced the phosphorylation of Y-box binding protein 1 (YB-1) promotes the repair of DNA-DSB and postirradiation survival through PI3K/AKT and MAPK/ERK signaling pathways in breast cancer [32]. This evidence concerns the gene AKT1 and cervical cancer.