In addition, miR-200 and miR-30 overexpression in anaplastic thyroid cancer induced high E-cadherin levels as well as the downregulation of vimentin, N-cadherin, ZEB1/2, SMAD2 and TFGBR1, in addition to the inhibition of cell invasion, highlighting their role as possible MET and differentiation regulators [33]. This evidence concerns the gene MET and thyroid gland undifferentiated (anaplastic) carcinoma.