Moreover, recently it was demonstrated that the treatment with the non-toxic natural agonist 2-(1′H-indole-3′-carbonyl)-thiazole-4-carboxylic acid methyl ester (ITE) of the ligand-activated transcription factor aryl hydrocarbon receptor (AhR), which is widely investigated as a promising anti-cancer drug target, interferes with the Jagged1-dependent Notch pathway activation and counteracts proliferation, invasion, and migration of TNBC cells [373,374]. Here, AHR is linked to cancer.