IL6 and acute respiratory distress syndrome: However, over the past decades, although great achievement has been made in the field of understanding the pathogenesis of ARDS/ALI, such as inositol could improve the surfactant functions and reduce IL-6 levels [45] or targeting IL-6 and other cytokine-induced “cytokine storm” [46] to alleviate ARDS, little convincing therapeutic strategies or agents are available to fully control the excessive inflammation until now.