Therefore, we investigated how stimulation or inhibition of non-canonical NF-κB signaling affects MuSCs from mdx mice, a mouse model for Duchenne muscular dystrophy, often used as a model for continuous muscle regeneration (Dangain and Vrbova, 1984; Anderson et al., 1987; Figure 5A). Here, NFKB1 is linked to Duchenne muscular dystrophy.