In addition, BRG1 is reported as a pro-oncogenic element in neuroblastoma, where BRG1 regulates multiple cellular processes, including cell growth, cell proliferation, cell cycle, and cell survival through oncogenic pathways such as BCL2 and PI3K/AKT (194), and BRG1 promotes breast cancer cell proliferation through multiple mechanisms, including fatty acid and lipid synthesis pathways, and ATP-binding cassette (ABC) transporter expression (192, 193). This evidence concerns the gene SMARCA4 and neuroblastoma.