The top significantly enriched terms were cytokine-mediated signaling pathway [18], response to interferon-gamma [19], peptidyl-tyrosine autophosphorylation, measles, JAK-STAT signaling pathway [20], cytokine-cytokine receptor interaction [21], Th17 cell differentiation [22], etc. Pai and Rodrigues explain the indirect signs of MTB exposure by interferon-gamma release assay, indicating that there is a cellular immune response to MTB [23]. The gene discussed is IFNG; the disease is measles.